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Search results “Determining intravascular vs extravascular hemolysis emedicine”
QUICK INTERNAL MED: Extravascular Vs Intravascular Hemolysis
 
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Views: 14334 allornonelaw
Sickle cell anemia - causes, symptoms, diagnosis, treatment & pathology
 
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What is sickle cell anemia? Sickle cell anemia is an autosomal recessive genetic condition where the beta-globin protein subunit of hemoglobin is misshapen, which ultimately leads to a sickle-shaped red blood cell, which is prematurely destroyed and can get stuck in small blood vessels. Find more videos at http://osms.it/more. Hundreds of thousands of current & future clinicians learn by Osmosis. We have unparalleled tools and materials to prepare you to succeed in school, on board exams, and as a future clinician. Sign up for a free trial at http://osms.it/more. Subscribe to our Youtube channel at http://osms.it/subscribe. Get early access to our upcoming video releases, practice questions, giveaways, and more when you follow us on social media: Facebook: http://osms.it/facebook Twitter: http://osms.it/twitter Instagram: http://osms.it/instagram Our Vision: Everyone who cares for someone will learn by Osmosis. Our Mission: To empower the world’s clinicians and caregivers with the best learning experience possible. Learn more here: http://osms.it/mission Medical disclaimer: Knowledge Diffusion Inc (DBA Osmosis) does not provide medical advice. Osmosis and the content available on Osmosis's properties (Osmosis.org, YouTube, and other channels) do not provide a diagnosis or other recommendation for treatment and are not a substitute for the professional judgment of a healthcare professional in diagnosis and treatment of any person or animal. The determination of the need for medical services and the types of healthcare to be provided to a patient are decisions that should be made only by a physician or other licensed health care provider. Always seek the advice of a physician or other qualified healthcare provider with any questions you have regarding a medical condition.
Views: 503686 Osmosis
Autoimmune Hemolytic Anemia | My Story
 
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More information on Autoimmune Hemolytic Anemia: https://rarediseases.info.nih.gov/diseases/5870/autoimmune-hemolytic-anemia → Shop Live Hope Lupus Merch (I appreciate your support) https://www.etsy.com/shop/livehopelupus → Check out my Amazon Storefront! (this is an affiliate link) Products that have helped me & products I love! https://www.amazon.com/shop/livehopelupus -------------------------- Follow me on Social Media: ↪ Instagram: @samanthamwayne ↪ Twitter: @samanthamwayne ↪ Facebook: www.facebook.com/livehopelupus ↪ Website: www.livehopefitness.com -------------------------- BUSINESS INQUIRES ONLY: [email protected] -------------------------- My name is Samantha Wayne and I have been creating advocacy videos on YouTube since 2009. I created Live Hope Lupus in order to create a space where those with chronic illnesses could get information and support. Please subscribe to follow me along on this journey! #hemolyticanemia #lupus #jaundice
Views: 2148 Live Hope Lupus
ANAEMIA causes,classification,clinical features,treatment
 
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Anaemia is a decrease in the RBC count, HGB and/or HCT values as compared to normal reference range for age and sex. Functionally defined as the inability of haemoglobin to supply the tissues with adequate oxygen.....hypoxia. ‘True’ anemia….decreased RBC mass and normal plasma volume. Pseudo or dilutional anemia….normal RBC mass and increased plasma volume . An increase in plasma volume may cause a dilutional or pseudo anemia (with low Hgb & HCT values) even though the RBC mass is normal ....can occur during pregnancy or caused by volume overload (IVs), congestive heart failure. 1. Classification of Anemia I. Etiologic Classification • Impaired RBC production • Excessive destruction • Blood loss II. Morphologic Classification • Macrocytic anemia • Microcytic hypochromic anemia • Normochromic normocytic anemia 2. Impaired RBC Production 1. Abnormal bone marrow • Aplastic anemia • Myelophthisis : Myelofibrosis, Leukemia, Cancer metastasis 2. Essential factors deficiency • Deficiency anemia : Fe, Vit. B12, Folic acid, etc • Anemia in renal disease : Erythropoietin 3. Stimulation factor deficiency • Anemia in chronic disease • Anemia in hypopituitarism • Anemia in hypothyroidism 3. Excessive Destruction of RBC Hemolytic anemia Intracorpuscular defect ;- • Membrane : Hereditary spherocytosis Hereditary ovalocytosis, etc. • Enzyme : G-6PD deficiency, PK def., etc. • Hemoglobin : Thalassemia, Hemoglobinopathies Extracorpuscular defect • Mechanical : March hemolytic anemia MAHA (Microangiopathic HA) • Chemical/Physical • Infection : Clostridium tetani • Antibodies : HTR, SLE • Hypersplenism Blood Loss • Acute blood loss : Accident, GI bleeding • Chronic blood loss : Hypermenorrhea, Parasitic infestation II. Morphologic Classification Macrocytic AnemiaMegaloblastic dyspoiesis • Vit. B12 deficiency: Pernicious anemia • Folic acid deficiency: Nutritional megaloblastic anemia, Sprue, Other malabsorption • Inborn errors of metabolism: Orotic aciduria, etc. • Abnormal DNA synthesis: Chemotherapy, Anticonvulsant, Oral contraceptives Microcytic Hypochromic AnemiaFe deficiency anemia: Chronic blood loss, Inadequate diet, Malabsorption, Increased demand, etc. • Abnormal globin synthesis: Thalassemia with or without Hemoglobinopathies • Abnormal porphyrin and heme synthesis: Pyridoxine responsive anemia, etc. • Other abnormal Fe metabolism: Normocytic Normochromic Anemia Blood loss • Increased plasma volume: Pregnancy, Overhydration • Hemolytic anemia: depend on each cause • Hypoplastic marrow: Aplastic anemia, RBC aplasia • Infiltrate BM: Leukemia, Multiple myeloma, Myelofibrosis, etc. • Abnormal endocrine: Hypothyroidism, Adrenal insufficiency, etc. • Kidney disease/Liver disease/Cirrhosis Hemolytic Anemia What is Hemolysis • Premature destruction of red cells. • Caused by hereditary and acquired disorders. Hemolysis occurs at two sites: Intravascular • Hemolysis occurs within systemic circulation. • Hemoglobin is released into plasma. • Hemoglobin is lost through kidneys or catabolized in the liver. Extravascular • Trapping of red cells in spleen or liver sinuses. • Lyses of trapped red cells. • Release of lysed hemoglobin and catabolism within the sequestering organ. Classification of Hereditary Hemolytic Anemia • Based on side effect: • Metabolic defect • Membrane defect • Hemoglobin defect A) Metabolic defect: • Defect in hexose monophosphate shunt: G-6-PD deficiency. • Defects of glycolysis: pyruvate kinase def., glucose phosphate isomerase deficiency. • Defects in red cell nucleotide metabolism: pyrimidine-5-nucleotidase def. B) Membrane defect: • Hereditary spherocytosis • Hereditary elliptocytosis • Hereditary pyropoikilocytosis C) Hemoglobin defect:  Thalassemias  Sickle cell anemia  Hemoglobin C disease  Hemoglobin E disease  Unstable hemoglobin
Views: 435 EAZZYMED
Anemia (Types, Lab Findings, High Yield Images)
 
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Views: 100902 DirtyUSMLE
Microangiopathic hemolytic anemia
 
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In medicine microangiopathic hemolytic anemia is a microangiopathic subgroup of hemolytic anemia caused by factors in the small blood vessels. It is identified by the finding of anemia and schistocytes on microscopy of the blood film. This video is targeted to blind users. Attribution: Article text available under CC-BY-SA Creative Commons image source in video
Views: 3255 Audiopedia
Rational Approach to Evaluation of Thrombotic Microangiopathy
 
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C. Christopher Hook , M.D., discusses the rational approach to the evaluation of thrombotic mircoangiopathy as part of the Mayo Medical Laboratories symposium "Coagulation Testing: What a Hematologist Should Know"
Hemolytic Anemia ¦ Treatment and Symptoms
 
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http://t.co/VyIFjqRj5x ◄ Hemolytic Anemia treatment — Finding the right information about Hemolytic Anemia treatment & symptoms, is crucial to managing Hemolytic Anemia. Learn more about the best current treatment for you. ► http://t.co/VyIFjqRj5x Website: http://www.MainMD.com Provides valuable health information, tools for managing your health, and support to those who seek information. You can trust that our content is timely and credible. Discussions: http://www.MainMD.com/forum YouTube: http://www.YouTube.com/MainMDcom Facebook: http://www.Facebook.com/MainMD Twitter: http://www.Twitter.com/MainMD THIS VIDEO CAN NOT BE RE-UPLOADED OR USED IN ANY WAY WITHOUT WRITTEN PERMISSION FROM MAINMD PRODS -MainMD © All Rights Reserved-
Views: 4281 MainMD
Anemia | Microcytic vs. Normocytic vs. Macrocytic
 
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Lesson on Anemia: Diagnosis and Causes. Anemia is defined as a decrease in RBC mass, with reduced hemoglobin and/or hematocrit. There are a many various causes of Anemia, and looking at the mean corpuscular volume (MCV) is helpful in determining the cause. Low MCV anemia is defined as microcytic anemia and has various causes including iron deficiency. Normal MCV anemia is defined as normocytic anemia with a variety of causes including hemolysis and bleeding and macrocytic anemia (high MCV) can be due to B12 deficiency or alcoholism.
Views: 645 JJ Medicine
Autoimmune Hemolytic Anemia; warm autoantibodies
 
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► Right Now! You can get access to all my hand-written hematology video notes (the notes that I use on my videos) on Patreon...There is a direct link through which you can view, download, print and enjoy! Go to https://www.patreon.com/medicosis Please subscribe to get new medical videos every week. My FAVORITE BOOK to study Pathology and pathophysiology for step 1 USMLE is Goljan, you can check it out here: https://goo.gl/9ojg6M My favorite book for internal medicine is Harrison's, check it out here: http://amzn.to/2A7UEdj My favorite book for surgery is this one, which contains notes and vignettes of Dr. Pestana, here: https://goo.gl/UdDGHq My favorite book for Ob.GYN is Hacker and Moore: https://goo.gl/umFsjc For a review book for USMLE step 1, check First Aid here: http://amzn.to/2zOU7w2 For a review book to study USMLE step 2CK, I recommend: http://amzn.to/2iI6zue For a review book to study USMLE step 2CS, check http://amzn.to/2z24RtR Check this pharmacology book, really easy to read, and inhexpensive: Pharmacology made incredibly easy: https://goo.gl/or3DzB If you would like a good pharmacology textbook, try Lippincott illustrated: https://goo.gl/PYEUP2 (Disclaimer: The medical information contained herein is intended for physician medical licensing exam review purposes only, and are not intended for diagnosis of any illness. If you think you may be suffering from any medical condition, you should consult your physician or seek immediate medical attention.) Disclaimer: these links are affiliate links, so that I get paid a percentage of the sale, to support the channel, however, the customer is not affected as they pay the same price. Click the Subscribe button to get new videos every week. To watch all my Hematology videos, check out this playlist: https://www.youtube.com/playlist?list=PLYcLrRDaR8_eoNz6dxXolh1XMEietcniU Like my page on Facebook: https://www.facebook.com/medicosis/ Follow us on twitter: https://www.twitter.com/medicosis Follow us on instagram here: https://www.instagram.com/medicosisperfectionalis/ ----My website will launch next year, so stay tuned… My dream is to do white board classroom teaching, audio podcast and live online webinars. But, I need your support to do this. To help support me and keep my videos available on youtube, visit me on Patreon,https://www.patreon.com/medicosis The funniest thing is that I have a playlist containing only mnemonics, yes, MEDICAL MNEMONICS, here: https://www.youtube.com/playlist?list=PLYcLrRDaR8_c4yXU3bwjIUNUk7C684ngd Whether you’re studying for the USMLE, shelf exam, COMLEX, PLEB, MCCEE, AMC CAT, these videos will help!
Serum Sickness - causes, symptoms, diagnosis, treatment, pathology
 
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What is serum sickness? serum sickness is a type III hypersensitivity reaction where foreign blood serum causes an allergy-like response. Find more videos at http://osms.it/more. Hundreds of thousands of current & future clinicians learn by Osmosis. We have unparalleled tools and materials to prepare you to succeed in school, on board exams, and as a future clinician. Sign up for a free trial at http://osms.it/more. Subscribe to our Youtube channel at http://osms.it/subscribe. Get early access to our upcoming video releases, practice questions, giveaways, and more when you follow us on social media: Facebook: http://osms.it/facebook Twitter: http://osms.it/twitter Instagram: http://osms.it/instagram Our Vision: Everyone who cares for someone will learn by Osmosis. Our Mission: To empower the world’s clinicians and caregivers with the best learning experience possible. Learn more here: http://osms.it/mission Medical disclaimer: Knowledge Diffusion Inc (DBA Osmosis) does not provide medical advice. Osmosis and the content available on Osmosis's properties (Osmosis.org, YouTube, and other channels) do not provide a diagnosis or other recommendation for treatment and are not a substitute for the professional judgment of a healthcare professional in diagnosis and treatment of any person or animal. The determination of the need for medical services and the types of healthcare to be provided to a patient are decisions that should be made only by a physician or other licensed health care provider. Always seek the advice of a physician or other qualified healthcare provider with any questions you have regarding a medical condition.
Views: 52008 Osmosis
Neonatal Anemia, Bleeding and Screening .
 
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Dr Abeer El Saka
Views: 326 Emp Pediatrics
Haptoglobin
 
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Haptoglobin (abbreviated as Hp) is the protein that in humans is encoded by the HP gene. In blood plasma, haptoglobin binds free hemoglobin (Hb) released from erythrocytes with high affinity and thereby inhibits its oxidative activity. The haptoglobin-hemoglobin complex will then be removed by the reticuloendothelial system (mostly the spleen). In clinical settings, the haptoglobulin assay is used to screen for and monitor intravascular hemolytic anemia . In intravascular hemolysis, free hemoglobin will be released into circulation and hence haptoglobin will bind the hemoglobin. This causes a decline in haptoglobin levels. Conversely, in extravascular hemolysis the reticuloendothelial system, especially splenic monocytes, phagocytose the erythrocytes and hemoglobin is not released into circulation; serum haptoglobin levels are therefore normal. This video is targeted to blind users. Attribution: Article text available under CC-BY-SA Creative Commons image source in video
Views: 7452 Audiopedia
Bilirubin Metabolism
 
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https://www.facebook.com/ArmandoHasudungan Support me: http://www.patreon.com/armando Instagram: http://instagram.com/armandohasudungan Twitter: https://twitter.com/Armando71021105
Views: 559226 Armando Hasudungan
PNH, The Role of Complement in PNH, and The Potential of Inhibiting Complement C3
 
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The complement system is an essential and powerful part of our immune defense system. Its main function is to coordinate the destruction and removal of infectious organisms, known and pathogens, or damaged cells in our body that need to be replaced.1 The complement system may be activated by three different pathways - the alternative, classical and lectin pathways that all run through Complement C3, the central protein in the cascade.1 C3 serves as the master switch controlling all downstream effectors that ultimately cause destruction of the cell. In situations where the complement system becomes over activated, it results in immune attack, the destruction of healthy cells, and ultimately causes illness and damage to the body.1 Paroxysmal nocturnal hemoglobinuria, PNH, is a rare, acquired, potentially life-threatening blood disorder, characterized by chronic, complement-mediated destruction of red blood cells—hemolysis – and formation of life-threatening blood clots, also known as thrombosis.2,3 In PNH, the body’s stem cells acquire a gene mutation which results in the production of abnormal blood cells. These defective red blood cells, white blood cells and platelets, lack the connector, known as GPI, for two important surface proteins – CD55 and CD59 – that regulate complement activity.4 As a result, the complement protein C3 becomes unregulated, triggering all downstream effectors that ultimately cause destruction of blood cells.5,6 In intravascular hemolysis, the formation of the membrane attack complex, known as MAC, creates holes in red blood cells causing them to rupture inside blood vessels. At the same time, extravascular hemolysis is caused by C3b being deposited on the surface of defective red blood cells, tagging them for removal and resulting in their destruction in the liver and spleen. Inhibiting the C3 protein may prevent both intravascular and extravascular hemolysis.7 APL-2 is an investigational therapy designed to help restore normal complement activity and reduce inflammation. By targeting C3, the central protein that serves as the master switch of the complement cascade, APL-2 has the potential to block activation from any complement pathway AND to prevent both intravascular and extravascular hemolysis, reducing the risk of thrombosis and anemia and transfusion dependency in patients with PNH.7 Learn more about the role of the complement system in PNH and the benefits of C3 inhibition in the following video and at https://pnhstudy.com/ 1. Murphy K, Weaver C. Innate immunity: the first lines of defense. In: Janeway's Immunobiology. 9th ed. London, UK: Garland Science; 2016. 2. Paroxysmal nocturnal hemoglobinuria (PNH). The Sidney Kimmel Comprehensive Cancer Center Web site. https://www.hopkinsmedicine.org/kimmel_cancer_center/types_cancer/paroxysmal_nocturnal_hemoglobinuria_PNH Accessed May 15, 2018. 3. Besa EC. Paroxysmal nocturnal hemoglobinuria (PNH) MedScape 2017; https://emedicine.medscape.com/article/207468-overview. Accessed May 15, 2018. 4. Rosse WF, Ware RE. The molecular basis of paroxysmal nocturnal hemoglobinuria. Blood. 1995;86(9):3277-3286. 5. Hill A, Kelly RJ, Hillmen P. Thrombosis in paroxysmal nocturnal hemoglobinuria. Blood. 2013;121(25):4985-4996; quiz 5105. 6. Parker C, Omine M, Richards S, et al. Diagnosis and management of paroxysmal nocturnal hemoglobinuria. Blood. 2005;106(12):3699-3709. 7. Data on file, Apellis Pharmaceuticals.
Reticulocytes
 
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► Right Now! You can get access to all my hand-written hematology video notes (the notes that I use on my videos) on Patreon...There is a direct link through which you can view, download, print and enjoy! Go to https://www.patreon.com/medicosis CORRECTION : I made a mistake, Reticulocytes have NO nucleus! The doubly corrected reticulocyte count is now called Reticulocyte production index. We will discuss the most important topics in hematology/ oncology! Watch all of my hematology videos here: https://www.youtube.com/playlist?list=PLYcLrRDaR8_eoNz6dxXolh1XMEietcniU
Back Home from the Hospital! Next Steps with Hemolytic Anemia & Lupus | Prednisone
 
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The coast isn’t quite clear yet but I’m happy I get to be home and resting. Thank you all for your positive thoughts and prayers. → Shop Live Hope Lupus Merch (I appreciate your support) https://www.etsy.com/shop/livehopelupus → Check out my Amazon Storefront! (this is an affiliate link) Products that have helped me & products I love! https://www.amazon.com/shop/livehopelupus -------------------------- Follow me on Social Media: ↪ Instagram: @samanthamwayne ↪ Twitter: @samanthamwayne ↪ Facebook: www.facebook.com/livehopelupus ↪ Website: www.livehopefitness.com -------------------------- BUSINESS INQUIRES ONLY: [email protected] -------------------------- My name is Samantha Wayne and I have been creating advocacy videos on YouTube since 2009. I created Live Hope Lupus in order to create a space where those with chronic illnesses could get information and support. Please subscribe to follow me along on this journey!
Views: 1308 Live Hope Lupus
Lower urinary tract infection (cystitis) - an Osmosis preview
 
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What is cystitis? Cystitis is any inflammation of the bladder. In the vast majority of cases, this inflammation is brought on by infection, meaning most cases of cystitis are actually a type of lower urinary tract infection. Find our complete video library only on Osmosis Prime: http://osms.it/more. Hundreds of thousands of current & future clinicians learn by Osmosis. We have unparalleled tools and materials to prepare you to succeed in school, on board exams, and as a future clinician. Sign up for a free trial at http://osms.it/more. Subscribe to our Youtube channel at http://osms.it/subscribe. Get early access to our upcoming video releases, practice questions, giveaways, and more when you follow us on social media: Facebook: http://osms.it/facebook Twitter: http://osms.it/twitter Instagram: http://osms.it/instagram Our Vision: Everyone who cares for someone will learn by Osmosis. Our Mission: To empower the world’s clinicians and caregivers with the best learning experience possible. Learn more here: http://osms.it/mission Medical disclaimer: Knowledge Diffusion Inc (DBA Osmosis) does not provide medical advice. Osmosis and the content available on Osmosis's properties (Osmosis.org, YouTube, and other channels) do not provide a diagnosis or other recommendation for treatment and are not a substitute for the professional judgment of a healthcare professional in diagnosis and treatment of any person or animal. The determination of the need for medical services and the types of healthcare to be provided to a patient are decisions that should be made only by a physician or other licensed health care provider. Always seek the advice of a physician or other qualified healthcare provider with any questions you have regarding a medical condition.
Views: 5471 Osmosis
Intravascular Coagulum
 
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Evacuation of trapped blood( Intravascular Coagulum) one week after hand sclerotherapy
Views: 174 SHAMMA Clinic
anemia mnemonic
 
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a simple code to remember various morphological types of anemia
Views: 5229 m tree
Blood groups and blood transfusion
 
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Hemolytic disease of the newborn, also known as hemolytic disease of the fetus and newborn, HDN, HDFN, or erythroblastosis fetalis,[1] is an alloimmune condition that develops in a peripartum fetus, when the IgG molecules (one of the five main types of antibodies) produced by the mother pass through the placenta. Among these antibodies are some which attack antigens on the red blood cells in the fetal circulation, breaking down and destroying the cells (hemolysis). The fetus can develop reticulocytosis and anemia. This fetal disease ranges from mild to very severe, and fetal death from heart failure (hydrops fetalis) can occur. When the disease is moderate or severe, many erythroblasts (immature red blood cells) are present in the fetal blood, and so these forms of the disease can be called erythroblastosis fetalis (or erythroblastosis foetalis). HDFN represents a breach of immune privilege for the fetus or some other form of impairment of the immune tolerance of pregnancy. Various types of HDFN are classified by which alloantigen provokes the response. In order of incidence, the types include ABO, anti-RhD, anti-RhE, anti-Rhc, anti-Rhe, anti-RhC, multiantigen combinations, and anti-Kell Signs and symptoms Signs of hemolytic disease of the newborn include a positive direct Coombs test (also called direct agglutination test), elevated cord bilirubin levels, and hemolytic anemia. It is possible for a newborn with this disease to have neutropenia and neonatal alloimmune thrombocytopenia as well. Hemolysis leads to elevated bilirubin levels. After delivery bilirubin is no longer cleared (via the placenta) from the neonate's blood and the symptoms of jaundice (yellowish skin and yellow discoloration of the whites of the eyes, or icterus) increase within 24 hours after birth. Like other forms of severe neonatal jaundice, there is the possibility of the neonate developing acute or chronic kernicterus, however the risk of kernicterus in HDN is higher because of the rapid and massive destruction of blood cells. It is important to note that isoimmunization is a risk factor for neurotoxicity and lowers the level at which kernicterus can occur. Untreated profound anemia can cause high-output heart failure, with pallor, enlarged liver and/or spleen, generalized swelling, and respiratory distress. HDN can be the cause of hydrops fetalis, an often-severe form of prenatal heart failure that causes fetal edema.[2] Complications Complications of HDN could include kernicterus, hepatosplenomegaly, inspissated (thickened or dried) bile syndrome and/or greenish staining of the teeth, hemolytic anemia and damage to the liver due to excess bilirubin. Similar conditions include acquired hemolytic anemia, congenital toxoplasma, congenital syphilis infection, congenital obstruction of the bile duct, and cytomegalovirus (CMV) infection. High at birth or rapidly rising bilirubin[3] Prolonged hyperbilirubinemia[3] Bilirubin Induced Neurological Dysfunction[4] Cerebral Palsy[5] Kernicterus[6] Neutropenia[7][8] Thrombocytopenia[7] Hemolytic anemia - Must NOT be treated with iron[9] Late onset anemia - Must NOT be treated with iron. Can persist up to 12 weeks after birth.[10][11][12] Pathophysiology Antibodies are produced when the body is exposed to an antigen foreign to the make-up of the body. If a mother is exposed to a foreign antigen and produces IgG (as opposed to IgM which does not cross the placenta), the IgG will target the antigen, if present in the fetus, and may affect it in utero and persist after delivery. The three most common models in which a woman becomes sensitized toward (i.e., produces IgG antibodies against) a particular antigen are hemorrhage, blood transfusion, and ABO incompatibility. Fetal-maternal hemorrhage, which is the movement of fetal blood cells across the placenta, can occur during abortion, ectopic pregnancy, childbirth, ruptures in the placenta during pregnancy (often caused by trauma), or medical procedures carried out during pregnancy that breach the uterine wall. In subsequent pregnancies, if there is a similar incompatibility in the fetus, these antibodies are
Views: 43 Medicos Adda
Aplastic Anaemia
 
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This is a Learning in 10 voice annotated presentation (VAP) on aplastic anaemia. To learn more about Learning in 10 (LIT), please visit learningin10.com. -- Learning in 10 (LIT) Reviews is a collection of 10-minute, user-friendly video lectures covering topics in the United States Medical Licensing Exam (USMLE) Step 2CK examination. LIT Reviews can be used by medical students to supplement their lecture materials. LIT Reviews have been created by world-class clinical faculty and each video undergoes a peer-review process to ensure accuracy of information.
Views: 572 Learning in 10
Autoagglutination  | autoagglutinated sample demo
 
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Autoagglutination represents clumping of an individual's red blood cells (RBCs or erythrocytes) by his or her own serum due to the RBCs being coated on their surface by antibodies. Autoagglutination is seen to occur in a type of immune mediated hemolytic anemia known as cold agglutinin disease. Here, the antibodies which act on the erythrocytes are active at temperatures below 30 degree C. Diagnosis of autoagglutination can be confirmed by a positive “Direct Antiglobulin Test” aka Direct Coomb’s test using Coomb’s serum (positivity is seen in 75% of cases) The development of the cold agglutinins (as cold reacting antibodies are also known as) may result from the following conditions -Infection with mycoplasma organisms -Infectious mononucleosis -Lymphoid malignant disorders -Unknown causes #Autoagglutination #Microhub
Views: 94 Microhub Plus
Haematopoesis and Myeloproliferative Disorder  - Overview
 
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Buy Images here: armandoh.org/shop "Myeloproliferative disorders are clonal stem disorders characterised by leukocytosis, thrombocytosis, erythrocytosis, splenomegaly, and bone marrow hypercellularity. Three classic disorders are described: primary myelofibrosis, essential thrombocythaemia and polycythaemia" Where do I get my information from: http://armandoh.org/resource Facebook: https://www.facebook.com/ArmandoHasudungan Support me: http://www.patreon.com/armando Instagram: http://instagram.com/armandohasudungan Twitter: https://twitter.com/Armando71021105 SPECIAL THANKS: Patreon members FaberCastell Australia - https://www.youtube.com/user/FaberCastellGroup What markers do I use? FaberCastellPITTartistpens1,5 FaberCastellPITTartistpensF FaberCastellPermanentmarkers FaberCastellPITTartistpensbrush
Views: 9584 Armando Hasudungan
Critical Care Paramedic 5:  Blood Administration
 
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This Wisconsin Critical Care Paramedic module covers blood administration as associated with critical care interfacility transports.
Views: 2218 WCTCEMS
Liver function tests
 
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Liver function tests are groups of clinical biochemistry laboratory blood assays designed to give information about the state of a patient's liver. The parameters measured include prothrombin time , aPTT, albumin, bilirubin , and others. Liver transaminases are useful biomarkers of liver injury in a patient with some degree of intact liver function. Most liver diseases cause only mild symptoms initially, but these diseases must be detected early. Hepatic involvement in some diseases can be of crucial importance. This testing is performed by a medical technologist on a patient's serum or plasma sample obtained by phlebotomy. Some tests are associated with functionality , some with cellular integrity , and some with conditions linked to the biliary tract . Several biochemical tests are useful in the evaluation and management of patients with hepatic dysfunction. These tests can be used to detect the presence of liver disease, distinguish among different types of liver disorders, gauge the extent of known liver damage, and follow the response to treatment. Some or all of these measurements are also carried out on those individuals taking certain medications — anticonvulsants are a notable example — to ensure the medications are not damaging the person's liver. This video targeted to blind users. Attribution: Article text available under CC-BY-SA
Views: 9367 encyclopediacc
ANEMIA (classification) : Hematology - Medical Lecture Review by Dr.Sadaf Alam MD Fellow
 
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There are too many lecture videos on each topic these days , finding the right lecture and right guidance to study a topic. Take help from this channel and save your time finding the right lecture in your preferred language. please check out other medical lecture reviews by dr.sadaf alam https://www.youtube.com/watch?v=VfGcw2eILwE&list=PL-4yUaugxOTjzrezp0bKW8HdDyuGWGMnY #NEET_PG#DNB#DOCTOR#MEDICAL#MEDSCHOOL#MEDICALLECTURE#MEDICAL VIDEO#VLOG#BLOG#HOWTOSTUDY#BEST#EASY#LECTURE#PATHOLOGY#PHARMACOLOGY#ANATOMY#SURGERY our need.
Views: 738 dr.sadaf alam
AGRANULOCYTOSIS AND NEUTROPENIA WHITE BLOOD CELL DEFICIENCIES
 
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Views: 1121 Health Apta
About pediatrics by dr.anand - smear picture of microcytic hypochromic picture
 
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Peripheral smear finding showing microcytic hypochromic anemia.
Anemia in the Elderly
 
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Views: 82 Jean G